Pathophysiology Over the last few decades, much has been discovered about the pathophysiological processes of chronic HF, especially myocardial remodeling, signaling important pharmacological targets that have proven to impact clinical outcomes. In the scenario of post-infarction HF, some peculiarities in these processes have been better understood, helping to understand specific characteristics of this clinical phenotype of HF and enabling a more targeted approach in this scenario. Initially, ischemia leads to loss of contractile function and consequently enlarges the ventricular cavity, […]
